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Narcotics, Steroids, AZT & Malnutrition cause AIDS
By Dr Mohammed Ali Al-Bayati, Ph.D., DABT, DABVT
Toxicologist and Pathologist (USA)
Review of the literature for the causes and the pathogenesis of AIDS worldwide revealed that approximately 90% of AIDS cases in the USA and Europe are observed in homosexual men and drug users. The regular uses of alcohol, heroin, cocaine, amphetamines, and alkyl nitrite cause chronic health problems in the nervous system, respiratory system, cardiovascular system, kidneys and other tissues in these individuals. The majority of these health problems are usually diagnosed currently as idiopathic, and treated with high doses of glucocorticoids and/or cytotoxic drugs. In addition, some homosexual men are usually heavy users of illicit drugs, alcohol, and oral and rectal glucocorticoids ( Fauci et al., 1998; Al-Bayati, 1999).

The treatment of a patient with prednisone at 60 mg per day for about three months can actually cause AIDS as described above. This treatment and doses are often given to patients suffering from lung fibrosis, thrombocytopenia, or other chemically induced chronic illnesses.

The reversal of CD4+ T-cells depletion in the peripheral blood was reported in HIV+ homosexual men after the termination of their treatment with glucocorticoids.

The medical evidence shows that AIDS is probably caused by the use of illicit drugs, therapeutic use of corticosteroids, and malnutrition. Differential diagnosis indicated that HIV is not involved.

There are thousands of cases infected with the HIV and they did not show any symptoms for more than 10 years (Fauci, et al., 1998; Al-Bayati, 1999).

Causes and pathogenesis of AIDS in drug users and homosexuals

The appearance of AIDS in the USA and Europe in drug users and homosexuals in the late 1970ís and early 1980ís coincided with the synergistic actions of several events. Briefly, these include the spread of illicit drug use, especially smoking crack cocaine and heroin in 1970ís, the approval of aerosols containing glucocorticoids by the U.S. FDA in 1976, the wide use of the glucocorticoid inhalers to treat chronic respiratory illnesses resulting from inhaling cocaine and heroin, the wide use of alkyl nitrites by homosexuals to facilitate anal sex in 1970ís, and the wide use of corticosteroids to treat chronic gastrointestinal tract illness in homosexuals. Furthermore, the approval of anti-retroviral drugs (AZT and protease inhibitors) and the steroids by the U.S. FDA to treat patients with AIDS and asymptomatic patients infected with HIV has exacerbated the problem (Al-Bayati, 1999).

The HIV hypothesis states that HIV causes AIDS by killing the CD4+ T-cells directly or indirectly (Fauci, et al., 1998). It appears that there is no scientific evidence to show that HIV can kill infected T4 cells (CD4+ T-cells) in vitro or in vivo. In addition, the abnormalities in the immune system of patients with AIDS are not restricted to the reduction of T4 cells as predicted by the HIV hypothesis. Hoxie et al. (1985) observed no evidence of death in T-cells infected with HIV in tissue culture. These cells continued to produce virus particles for more than four months after inoculation with the virus. Many reports describe the changes in the lymph nodes of patients infected with HIV and these changes range from extensive cellular hyperplasia (excessive production) of T and B lymphocytes and the supporting stroma to severe atrophy of the glands. Changes in the lymph nodes of 505 HIV- infected patients who were asymptomatic or had AIDS demonstrate three distinct stages. These are hyperplasia (245 patients), atrophy (117 patients), and mixed stage (172 patients) (Al-Bayati, 1999). The occurrence of hyperplasia in the infected lymph nodes contradicts the HIV hypothesis which states that HIV destroys infected T-cells (Gallo, 1987; Fauci et al., 1998).

The changes in the lymph nodes described above are not unique to HIV-infected individuals, but have also been found in HIV-negative patients in risk groups. The lymph nodes of 215 HIV-negative homosexual men who were drug users showed hyperplasia and atrophy, and lymph nodes of 15 men showed Kaposiís sarcoma and lymphoma. These changes are AIDS-indicator diseases based on the CDCís criteria, yet the subjects were HIV-negative (Al-Bayati, 1999).

Duesberg, (1992) stated that HIV infects on the average only 0.1% (1 out of 500 to 3000) of T-cells in AIDS patients, and at least 3% of all T-cells are regenerated during the two days it takes a retrovirus to infect a cell. HIV could never kill enough T-cells to cause immunodeficiency. Thus, even if HIV killed every infected T-cell, it could deplete T-cells only at 1/30 of their normal rate of regeneration, not considering activated regeneration. The study by me and my colleagues (Al-Bayati et al. 1990) also showed that the rate of regeneration in the damaged thymus and lymphoid tissue of mice treated with a lymphotoxic agent (vanadate) was very rapid.

In addition to illicit drug and alcohol abuse, some homosexuals are also heavy users of alkyl nitrites that relax the anal muscle and facilitate anal sex. It has been stated that the use of alkyl nitrites permeated the gay life by 1977 (Al-Bayati, 1999).

Some homosexuals usually suffer from acute and chronic rectal and gastrointestinal diseases that dictate the heavy therapeutic use of rectal steroids. Among 7 selected studies that included 736 patients who were infected with HIV and/or had AIDS, 97% were homosexual or bisexual men. They show clearly that homosexual men suffer from extensive rectal and gastrointestinal problems that result in chronic use of therapeutic rectal steroids (Al-Bayati, 1999).

Review of the medical literature revealed that the short and the long term use of glucocorticoids at therapeutic doses, resulted in a variety of effects on the immune system that range from a transient reduction in T-cells count in peripheral blood to the development of full blown AIDS. Since the harmful effect of corticosteroids is broad, it is not surprising that many types of infections seem to occur more often in patients treated with corticosteroids.

In addition, Kaposiís sarcoma (KS) can develop, independently of HIV, in patients chronically treated with glucocorticoids. Many cases, which developed KS following treatment with glucocorticoids, had reversal of their lesions after the termination of the treatment (Al-Bayati, 1999).

The high prevalence of adrenal insufficiency among AIDS patients provides very strong evidence that AIDS in these patients is caused by the use of corticosteroids. The most common abnormality seen in HIV-infected individuals is hyponatremia, which is found in up to 30 percent of patients. Scientists have also stated that the presence of a low sodium level, combined with a high serum potassium level, in a patient should alert one to the possibility of adrenal insufficiency and adrenocortical insufficiency as seen following prolonged administration of excess glucocorticoids.

Furthermore, as stated above, that the CD4+ T-cells depletion in the peripheral blood of HIV-positive homosexual men was reversed after the termination of their treatment with glucocorticoids, and at least 77% of  2349 patients who participated in the four major AZT clinical trials (1987-1992) were HIV-negative prior to their treatment with AZT. These studies demonstrate clearly that HIV is not the cause of AIDS (Al-Bayati, 1999).

Causes and pathogenesis of AIDS in infants
and children in USA and Europe
As of January 1, 1997, the number of infants and children in USA diagnosed with AIDS was  6891, and ninety percent of these cases had mothers who were drug users (Fauci et al., 1998; Al-Bayati, 1999). The results of nine large studies surveying the prevalence of drug use in relation to the outcome of pregnancy in the USA showed that up to 15% of pregnant women used cocaine during pregnancy based on a positive urine test.

Infants and children with AIDS are dying from opportunistic infections as a result of malnutrition and the excessive use of therapeutic steroids to treat the wide range of illnesses in these children.

Causes and pathogenesis of AIDS in hemophiliac

The medical evidence suggests that AIDS in hemophiliac patients is probably caused by the treatment with immunosuppressive agents (cyclophosphamide and glucocorticoids) which have been used to prevent the development of antibodies to factors VIII and XI in patients with hemophilia (Al-Bayati, 1999). Patients with severe hemophilia have serious chronic joint problems resulting from bleeding inside the joints. That is also treated with glucocorticoids (Al-Bayati, 1999). AIDS has been reported in HIV-negative and HIV-positive hemophiliac patients.

Causes and pathogenesis of AIDS in organ transplant
and / or blood transfusion patients
As of January 1, 1997, the number of patients who received blood transfusions, blood components or tissues, and then subsequently developed AIDS in USA is  7888 (Al-Bayati, 1999). Furthermore, glucocorticoids and other immuno-suppressive agents are also used to prevent tissue rejection in organ transplant patients.

Blood transfusion patients and people with organ transplants develop AIDS after being treated with corticosteroids, and the list of opportunistic diseases described in these groups are similar to those described in people with AIDS (Fauci, et al., 1998).

Causes and pathogenesis of AIDS in Africa

There is extensive literature describing the impact of malnutrition on the function and the structure of the immune system in people in Africa (Al-Bayati, 1999). This information clearly demonstrates that AIDS in Africa is more likely to be caused by starvation than by HIV. The functions of the immune system, especially the cellular immunity, are impaired in malnutrition cases. The severity of the impairment is dependent on the degree of malnutrition in both humans and animals.

Atrophy in the lymphoid organs in malnourished people is caused by increased levels of cortisol as well as by protein and vitamin deficiency. The reduction in the thymus and the lymphoid tissue size and the reduction in the function of the immune system of malnourished children and animals were reversed after proper feeding. For example, the size of the thymus increased from 20% of normal in a malnourished child to 107% of normal following 9 weeks of proper feeding. The reversal in the reduction of CD4+ T-cell count in HIV+ pregnant women following proper feeding was also reported by Fawzi et al., (1998). Briefly, the influence of diet on T-cell counts in peripheral blood was studied in 1075 HIV-infected pregnant women who had poor nutritional status. The CD4+ T-cell counts of the women who received multivitamins increased from 424/ĶL to 596/ĶL during six months of proper feeding.

Furthermore, the large study of Fawzi et al., 1998, clearly demonstrated that HIV is not implicated, and the impairment of the immune system in a mother (HIV-positive) who suffers from malnutrition can be reversed by feeding the mother proper nutrition. This treatment also improved the outcome of pregnancy.


The logical steps that should be taken to prevent AIDS and to cure people with AIDS are:
1) Prevent the causes of AIDS by educating the public about the toxic effects of the illicit drugs and alcohol.
2) Limit the use of glucocorticoids in the treatment of chronic conditions and in the treatment of people with AIDS. 

3) Monitor the levels of CD4+ T-cells and CD8+ T-cells in the blood of patients who are receiving medium or high therapeutic doses of glucocorticoids for significant periods. 

4) Discontinue the treatment of patients having AIDS and asymptomatic patients with AZT and protease inhibitors immediately. These are very toxic medications. 

5) Provide proper clinical support and nutrition to patients with AIDS based on their medical needs. Prior to the development of full-blown AIDS in drug users and homosexuals, the damage is caused by the use of drugs.

6) Alpha lipoic acid is a powerful antioxidant that has been used to prevent injury caused by chemicals in vivo and in vitro and injuries in diabetic patients for the last two decades (Al-Bayati, 1999). It has been used in Europe to reverse peripheral neuropathy in diabetic patients and has been shown to be effective and safe in several clinical trials. This drug is very effective in preventing and reversing injuries resulting from metabolic changes and/or exposure to chemicals that induce lipid peroxidation. This medication should be given to people with AIDS to boost the immune system and to heal tissue injury (Al-Bayati, 1999).

Al-Bayati, M.A. (1999). Get All The Facts: HIV does not Cause AIDS. Toxi-health    
International, Dixon, California (http://www.toxi-health.com)

Al-Bayati, M.A.; Culbertson, R.M.; Schreider, J.P.; Rosenblatt, L.S.; and Raabe, O.G. (1992). The Lymphotoxic Action of Vanadate. Journal of Environmental Toxicology and Oncology, 11(2):19-27.

Duesberg, P.H. (1992). AIDS Acquired by drug consumption and other noncontagious Risk Factors. Pharmac. Ther.  Vol.55, 201-277.  

Fauci A.S.; Braunwald, E.; Isslbacher, K.J.; et al. Harrisonís.  Principles of Internal Medicine.  (McGraw-Hill  Companies, Inc. New York USA, ed. 14, 1998).

Fawzi, W.W.; Msamanga, G.I.; Spiegelman, D.; et al. (1998). Randomized trial effects of vitamin supplements on pregnancy outcomes and T cell counts in HIV-1-infected women in Tanzania. The Lancet 351:1447-1482.

Gallo, R.C. (1987). The AIDS Virus. Scientific America. 256:46-56.

Hoxie, J.A.; Haggarty, B.S.; Rackowski, J.L.; et al. (1985). Persistent Noncytopathic Infection of  Normal Human T lymphocytes with AIDS-Associated Retrovirus. Science 229(4720):1400. 

Dr Mohammed Ali Al-Bayati
Ph.D., D.A.B.T., D.A.B.V.T.
Toxicologist and Pathologist
Toxi-Health International
150 Bloom Dr.
Dixon, CA 95620

Tel: (707) 678-4484
Fax: (707) 678-8505

For more articles and information about Dr Mohammed Ali Al-Bayati visit the following website:
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